2017
Using Pseudomonas syringae effectors to discover and manipulate soybean immunity components
Contributor/Checkoff:
Category:
Sustainable Production
Keywords:
GeneticsGenomics
Lead Principal Investigator:
James R Alfano, University of Nebraska
Co-Principal Investigators:
Project Code:
1717
Contributing Organization (Checkoff):
Institution Funded:
Brief Project Summary:

The bacterial pathogen Pseudomonas syringae pv. glycinea (Pgl) causes the economically significant bacterial blight disease of
soybean. One of the primary pathogenic strategies that P. syringae uses is the injection of type III effector proteins into plant cells.
The majority of these proteins act to suppress plant immunity. The potential to elucidate their target proteins and sites of action
makes type III effectors important molecular tools for finding new components of innate immunity that may not be identifiable using
other approaches. Because many of these components are likely targeted by effectors from oomycete and fungal plant pathogens as
well, their identification may also...

Unique Keywords:
#breeding & genetics
Information And Results
Project Deliverables

i. Complete the experiments to determine if the soybean GRP7 RNA-binding protein can be
manipulated to improve resistance to biotic stress in the field.
ii. Continue to identify Pseudomonas syringae pv. glycinea LN10 type III effectors soybean targets
using multiple approaches.
iii. Determine if the microtubule network, a target of multiple type III effectors, can be manipulated to
improve plant immunity.

Final Project Results

Updated April 24, 2018:

View uploaded report PDF file

The United Soybean Research Retention policy will display final reports with the project once completed but working files will be purged after three years. And financial information after seven years. All pertinent information is in the final report or if you want more information, please contact the project lead at your state soybean organization or principal investigator listed on the project.